Cialis works by inhibiting phosphodiesterase-5 (PDE5), a specific enzyme. This inhibition increases levels of cyclic guanosine monophosphate (cGMP), a crucial molecule for smooth muscle relaxation.
Specifically:
- PDE5 normally breaks down cGMP. By blocking PDE5, Cialis allows cGMP to persist. Increased cGMP leads to relaxation of smooth muscles in the blood vessels of the penis. This relaxation improves blood flow, facilitating an erection.
This mechanism differs from other medications. It’s a targeted approach, focusing on a specific enzyme involved in vascular function.
Importantly, Tadalafil’s effect on PDE5 is relatively long-lasting, contributing to its longer duration of action compared to other drugs in its class. The half-life of Tadalafil is approximately 17.5 hours.
However, individual responses to Cialis vary. Factors like age, overall health, and other medications can influence its effects. Consult your doctor to discuss potential interactions and side effects.
Remember that this information provides a general overview. Detailed medical advice should always come from a healthcare professional.
